Chapter 1 Chairman's creation (pages 1–3): Colin F. Mills
Chapter 2 nutritional assets of Copper (pages 5–22): H.T. Delves
Chapter three Absorption, delivery and Distribution of Copper (pages 23–48): Ian Bremner
Chapter four Metabolic Interactions of Copper with different hint parts (pages 49–65): Colin F. Mills
Chapter five Copper Proteins and Copper Enzymes (pages 71–91): A.E.G. Cass and H.A.O. Hill
Chapter 6 Caeruloplasmin: A Multi?Functional Metalloprotein of Vertebrate Plasma (pages 93–124): Earl Frieden
Chapter 7 Superoxide Dismutases (pages 125–142): Hosni Moustafa Hassan
Chapter eight Copper, Biogenic Amines, and Amine Oxidases (pages 143–161): T.L. Sourkes
Chapter nine Copper and the Synthesis of Elastin and Collagen (pages 163–182): Edward D. Harris, John okay. Rayton, James E. Balthrop, Robert A. Di Silvestro and Margaret Garcia?de?Quevedo
Chapter 10 Copper Deficiency in Ruminants (pages 183–207): Cecil H. McMurray
Chapter eleven Copper Deficiency in people (pages 209–225): David M. Danks
Chapter 12 Copper in Fetal and Neonatal improvement (pages 227–245): Lucille S. Hurley, Carl L. willing and Bo Lonnerdal
Chapter thirteen Copper and Neurological functionality (pages 247–266): D.M. Hunt
Chapter 14 Copper and Hepatic functionality (pages 267–282): Charles A. Owen
Chapter 15 healing makes use of of Copper?Chelating brokers (pages 283–299): Ragnar Osterberg
Chapter sixteen Anaerobic Potentiation of Copper Toxicity and a few Environmental issues (pages 301–324): David C.H. McBrien
Chapter 17 Chairman's ultimate feedback (pages 325–330): Colin F. generators
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Additional info for Ciba Foundation Symposium 79 - Biological Roles of Copper
E. a mixed-metal) soluble form? Riordan: Both the soluble and insoluble forms contain mixed metals but the soluble form predominantly contains zinc whereas the insoluble form predominantly contains copper. Mills: I am thinking of the problems that paediatricians face because of perinatal accumulation of copper. Most of the copper is, presumably, in the insoluble polymeric form. This balance between the soluble and the insoluble forms may be determined in some way by zinc status. There must be marked differences in turnover between the soluble and insoluble forms.
I suspect that we may have been wrong to attribute the ABSORPTION, TRANSPORT AND DISTRIBUTION OF COPPER 41 disappearance of [35S]cysteinefrom metallothionein in the liver of copperinjected rats to the degradation of the protein (Bremner et a1 1978). Conceivably, we were actually looking at the consequences of aggregation of the protein, which would be consistent with what we know of the tendency of copper-metallothionein to form polymeric species. We are currently examining this possibility. e.
Results are given as the proportion of the administered dose recovered in the ‘empty’ carcass, in the liver, kidneys and small intestinal mucosa four hours after the dose. Data for both control and zinc-supplemented rats ( PZZZA) are taken from Hall et a1 (1979). (a) ABSORPTION, TRANSPORT AND DISTRIBUTION OF COPPER 27 Similar observations have been made in cadmium-supplemented rats (Davies & Campbell 1977). 6 pg cadmium/g, was associated with an increase in the binding of mucosal @Cu to a low-molecular-weight protein, which was almost certainly metallothionein.