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Ciba Foundation Symposium 106 - Human Cataract Formation

Chapter 1 advent (pages 1–2): A. Spector
Chapter 2 class of Human Cataractous swap by way of the yank Cooperative Cataract study crew technique (pages 3–24): Leo T. Chylack
Chapter three Epidemiological and different experiences within the overview of things Contributing to Cataractogenesis (pages 25–47): R. M. Clayton, J. Cuthbert, J. Seth, C. I. Phillips, R. S. Bartholomew and J. Mck. Reid
Chapter four Oxidation and Cataract (pages 48–64): Abraham Spector
Chapter five Metabolism and serve as of Glutathione within the Lens (pages 65–87): Venkatn. Reddy and Frank J. Giblin
Chapter 6 Cataracts and Photochemical harm within the Lens (pages 88–109): Raymond F Borkman
Chapter 7 Diabetic and Galactosaemic Cataracts (pages 110–131): Peter F. Kador and Jin H. Kinoshita
Chapter eight Calcium and the body structure of Cataract (pages 132–162): George Duncan and Tim J. C. Jacob
Chapter nine Cytoskeletal Proteins of the growing older Human Lens (pages 163–176): Harry Maisel
Chapter 10 interplay of Crystallins with the Cytoskeletal–Plasma Membrane advanced of the Bovine Lens (pages 177–190): Hans Bloemendal, Wilfried W. De Jong, Frans C. S. Ramaekers, Alphons J. M. Vermorken, Irene Dunia and E. Lucio Benedetti
Chapter eleven Crystallin Genes: Templates for Lens Transparency (pages 191–207): Joram Piatigorsky, John M. Nickerson, Charles R. King, George Inana, J. Fielding Hejtmancik, James W. Hawkins, Teresa Borras, Toshimichi Shinohara, Graeme Wistow and Barbara Norman
Chapter 12 The Crystallin Gene households (pages 208–217): John G. G. Schoenmakers, Johan T. Den Dunnen, Rob J. M. Moormann, Rosalie Jongbloed, Rob W. Van Leen and Nicolette H. Lubsen
Chapter thirteen The Molecular buildings and Interactions of Bovine and Human ??Crystallins (pages 219–236): Lesley Summers, Christine Slingsby, Helen White, Michael Narebor, David Moss, Linda Miller, Daruka Mahadevan, Peter Lindley, Huub Driessen, Tom Blundell, Johan Den Dunnen, Rob Moormann, Rob Van Leen and John Schoenmakers
Chapter 14 The Molecular foundation of Cataract Formation (pages 237–247): George B. Benedek
Chapter 15 Non?Invasive strategies within the research of Cataract improvement on the Metabolic and Protein Molecular point (pages 248–274): William H. Garner

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There is evidence that heterozygous individuals with reduced rates of galactose clearance may develop presenile cataracts (Skalka et a1 1980); probably their galactose consumption also RISK FACTORS IN CATARACTOGENESIS 35 affects the rate of opacification in such individuals. Both erythrocytes and lens express glucose-6-phosphate dehydrogenase, and genetic variants which confer susceptibility to haemolysis under certain conditions may also confer a liability to cataract formation (Orzalesi et a1 1981).

Examples of data sheets are shown in our previous publications. ead of the household. The population was matched for age and sex (Clayton eit a1 1980, 1982, Bartholomew et a1 1980). , to be significantly different between the cataract and control populations are shown in Table 1. Overall differences between the populations are shown in Fig. 1. >- u z 2w 18 L1: 6 1 I in DISCRIMINANT SCORE FIG. 1. The two populations of patients and controls between the ages of 50 and 89 were compared by stepwise discriminant analysis using the SPSS procedure (Statlstical Package for the Social Sciences: see Nie et al 1975).

0001 0 . 06 NS 5 . 01 4 . 0001 CLAYTON E T A L 30 TABLE 2 Distribution of cataract and control populations by social group Patients Controls Social proup No. % No. 049. The x2 value shows slight mismatching, significant at the 5% level. While groups I, I1 and IIIM are matched, group IIIN has a very small excess of controls, and groups IV and V have a small excess of patients. The effect in the population comparisons is discussed in the text. These include calcium, cholesterol, total protein, albumin and fasting glucose levels.

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