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Asthma and Allergic Diseases: physiology, by Gianni Marone

By Gianni Marone

This publication updates the former assurance of the mechanisms and mediators of bronchial asthma and its therapy. It offers articles by way of the key names within the box.

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IL-1/3, TNF-c~, LPS or PMA) results in kinase activation and subsequent phosphorylation of the I-roB molecule, which is targeted for rapid degradation causing dissociation of the cytoplasmic NF-tcB-I-tcB complexes (Fig. 7) (7). Active NF-~cB heterodimers then translocate to the nucleus where they bind ~cB elements in responsive genes causing activation of transcription (7). I-~cB proteins can be divided into two distinct classes. The first consists of the precursor forms of the p50 and p52 subunits of NF-tcB.

Immunogenetics 26(4-5):230-236, 1987. Reid, M. , Nish, W. , Whisman, B. , Goetz, D. , Hylander, R. , Parker, W. , Jr and Freeman, T. M. HLA-DR4-associated nonresponsiveness to mountain cedar allergen. J. Allergy Clin. Immunol. 89:593-598, 1992. , Kemeny, D. , Welsh, K. I. and Lee, T. H. An HLA-associated nonresponsiveness to mellitin: a component of bee venom. J. Allergy Clin. Immunol. 86:160-170, 1990. , Muto, M. and Ohta, N. HLA-linked genes controlling immune response and disease susceptibility.

C. M. and Szczeklik, A. Aspirin-induced asthma and HLA-DRB 1 and HLA-DPB 1 genotypes. Clin. Exp. Allergy 27:574-577, 1997. Loveridge, J. , Rosenberg, W. M. , Kirkwood, T. B. L. and Bell, J. I. The genetic contribution to human T-cell receptor repertoire. Immunology 74:246-250, 1991. Moss, P. A. , Rosenberg, W. M. , Zintzaras, E. and Bell, J. I. Characterization of the human T cell receptor a-chain repertoire and demonstration of a genetic influence on V~ usage. Eur. J. Immunol. 23:1153-1159, 1993.

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